Soluble IL-2R & sarcoidosis
Some search terms matchedExplore sIL2R, sCD25, soluble interleukin-2 receptor, T-cell activation, and sarcoidosis.
THE PURSER WELLNESS ENCYCLOPEDIA
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What’s searchable?Explore sIL2R, sCD25, soluble interleukin-2 receptor, T-cell activation, and sarcoidosis.
Explore death receptor 4, TRAIL-R1, TRAIL, apoptosis, and programmed cell death.
Explore TNFSF10, TRAIL, DR4, DR5, apoptosis, immune surveillance, and cell-death signaling and treatment research.
What if your food isn't as nutritious as you think? Calories are easy to count. Protein is easy to track. But the real story lies in the vitamins, minerals, antioxidants, and trace nutrients your body depends on every single day. Most
What if the reason you still feel exhausted, inflamed, and stuck isn't a lack of supplements... but a lack of cellular energy? Most people focus on symptoms. Few ever address what’s happening inside the cell. CMA with REDOX is changing th
What if everything you’ve been told about PCOS is missing the real culprit? Homocysteine isn’t just a random lab marker—it may be the hidden thread tying together hormone imbalance, inflammation, and the symptoms so many are struggling to m
High homocysteine often shows up first as widespread inflammation—pain all over, poor circulation, or even early warning signs of cardiovascular problems like stroke and heart attack. The best way to know for sure is through testing: a blood test can measure homocysteine directly, while a CMA with Redox uncovers the vitamin deficiencies driving it. For genetic causes, the DRP Genetics panel screens 15–16 of the 17 known SNPs that can cause permanently high homocysteine. If your levels are fixed by genetics, reduced glutathione and targeted B vitamins (like MTHFR Daily) are key to balancing the
Searchable captionsRead matching passageThe real root of fibromyalgia? It’s not just pain — it’s copper. We’ve uncovered that fibromyalgia is often driven by genetic mutations (like CBS, GAD1, GAD2) that affect how your body handles copper. The copper builds up in your blood but never makes it into your cells — leading to widespread pain, inflammation, and migraines.
Searchable captionsRead matching passageStruggling with high blood pressure that just won’t budge—even with multiple meds? It might be due to genetic issues causing high homocysteine , which can stiffen your arteries through IL-8 and MCP-1 cytokines.
Did you know scurvy is still around today? It might sound like an old pirate disease, but a lack of Vitamin C can still cause fatigue, joint pain, bleeding gums, and slow healing. Without Vitamin C, your body can’t make collagen, which is essential for healthy skin, blood vessels, and connective tissues.
Scurvy, no one should have it in these modern ages, but I see these patients again and again. Thiamine Deficiency is another that NO ONE knows to look at, but it’s there. Remember, these are INTRACELLULAR deficiencies, not just blood serum levels. You have to Dig Deep to get the root of the issue.
High homocysteine levels can lead to inflammation, joint and muscle pain, and even shifting pain throughout the body. It’s also linked to birth defects and chronic kidney failure, and can raise blood pressure. Six genetic SNPs (like MTR, MTRR, and CBS) and MTHFR mutations (like C677T) can prevent homocysteine from converting into glutathione, which is essential for keeping inflammation in check. Plus, 12 vitamin deficiencies may temporarily raise homocysteine levels, so addressing these deficiencies is key.
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