Let’s explore “IL-6 & liver health”

MTHFR: THE TERRIBLE - but what is the problem I see with MTHFR SNPs? Inflammation caused by homocysteine! If you have high homocysteine, you get high inflammatory cytokine release. The only way to balance it out is reduced glutathione

High homocysteine levels can lead to inflammation, joint and muscle pain, and even shifting pain throughout the body. It’s also linked to birth defects and chronic kidney failure, and can raise blood pressure. Six genetic SNPs (like MTR, MT

Ever wondered how Homocysteine, MTHFR, and Glutathione are connected? Homocysteine is a highly inflammatory substance that triggers cytokine release in the body, fueling inflammation. It’s part of the methionine cycle, which links directly

A true functional glutathione would help prevent the cytokine cascade that leads to the cytokine storm with the invisible scourge. It would also help ameliorate the inflammatory response to the displaced heme iron we now know is being found

Ask your clinician to define genes/analytes, specimen, assay, repeatability, and how results change management.

Explore the possible links between homocysteine, immune signaling, and pain.

Separate questions about toxin exposure from questions about genetics and antioxidant pathways.

See how IL-8 and MCP-1 feature in research on kidney inflammation and scarring.

Explore why glutathione is studied in kidney disease without assuming a product improves kidney function.

Learn why an IL-8 result alone cannot locate an infection.