Let’s explore “Hashimoto’s thyroiditis and cytokines”

Dr. Purser shares his clinical perspective on homocysteine, methylation, and inflammatory patterns.

A sudden spike in folate or homocysteine is rarely caused by supplements. It is most often the result of a hidden infection or minor injury triggering a cytokine surge. Identifying and treating the underlying source is the key to bringing l

Remember all the talk about cytokine storms a few years back? 🌪️ These intense inflammatory responses are often triggered by overstimulated white blood cells—and low glutathione levels may be the reason why. If you’re dealing with lingering

MTHFR: THE TERRIBLE - but what is the problem I see with MTHFR SNPs? Inflammation caused by homocysteine! If you have high homocysteine, you get high inflammatory cytokine release. The only way to balance it out is reduced glutathione

High Homocysteine Levels and Inflammation: What You Need to Know High homocysteine levels are a common issue, often linked to genetics. There are seven gene variants (like MTHFR, CBS, and others) that can make it tough to bring those level

High homocysteine levels are a common issue, often linked to genetics. There are seven gene variants (like MTHFR, CBS, and others) that can make it tough to bring those levels down. If you're dealing with this, the key is to look at your IL

A true functional glutathione would help prevent the cytokine cascade that leads to the cytokine storm with the invisible scourge. It would also help ameliorate the inflammatory response to the displaced heme iron we now know is being found

See how IL-8 and MCP-1 feature in research on kidney inflammation and scarring.

Explore dimethylarginine dimethylaminohydrolase 1 (DDAH1), asymmetric dimethylarginine (ADMA), nitric oxide, blood vessels, and related blood-vessel pathways.

Explore RAD50, the MRE11–RAD50–NBS1 (MRN) complex, DNA double-strand breaks, genomic stability, and related DNA-repair and antioxidant pathways.