Let’s explore “Insulin receptor signaling”

Insulin Resistance, when properly diagnosed and treated, is reversible. You can FLIP out of it! But how? You need to get to the ROOT of the issue - dysfunctional insulin receptors on the cells. These receptors don’t have all they need to fu

What are the major contributors to insulin resistance? Zinc, Chromium, Omegas, B12, and Selenium. Check your intracellular levels and get these in check, watch that insulin resistance reverse quickly. Active lifestyle and eating good will m

The connection between Chromium and Insulin, swipe to learn! . . .

Is Type II reversible? Yes

11 - 15 of my TOP 20 Medical Truths, 13 may be of interest to you: 11. New-Onset TYPE 2 DM (diabetes mellitus)? Then low vitamins (zinc, chromium, b12) could be the cause affecting insulin receptor function. Check the intracellular vitami

This is another post for my FACEBOOK LIVE tonight at 7PM MST (in a little over 2 hours). This is imagery of an insulin receptor accepting insulin and then turning on a GLUT-4 glucose receptor (really a gateway) which allows glucose (crude

Omega-3 fish oils contain N-PUFAs which are like the motor oil for the little engines that are called insulin receptors. Without the oil they fail. Just like engines will do. Got it? See you in 2 hrs at me FACEBOOK LIVE on intracellular te

Dupilumab, sold as Dupixent, is a prescribed antibody medicine that acts on IL-4 receptor alpha.

HRH2 is the gene name for Histamine Receptor H2. Learn what a specific result could mean and what to ask about it.

IL23R encodes part of the receptor that receives IL-23 signals. This connects the gene to the maintenance of Th17-related immune responses, which differ from the Th2 patterns discussed in allergy.

…-6, a cytokine involved in immune responses and acute-phase signaling. It can appear in discussions of infection, chronic inflammation and metabolic health, with effects that depend on the context. Also called: IL-6, Interleukin 6 IL-6 link…

NR3C2 encodes the mineralocorticoid receptor, which responds to hormones such as aldosterone and helps regulate salt and fluid balance. It is distinct from the NR3C1 glucocorticoid receptor.