Let’s explore “COMT and catecholamine processing”

Explore what COMT does and how to read variant results with context.

Makes an enzyme that helps break down catechol compounds, including some neurotransmitters. A common variant does not by itself explain mood, attention or stress tolerance.

If you have a COMT mutation, your body may not break down estrogen or absorb B12 properly. 😵‍💫 The fix? Add DIM (like DIM Avail) for estrogen balance and use adenosyl + hydroxy B12 for better absorption. Start with drops or go straight to c

Can you take bioidentocal hormone replacement therapy if you have COMT?? Some say no, and they are wrong. You HAVE to use bioidential hormones and you HAVE to use someone that knows what they’re doing for treatment. If not, you may have p

Well, let’s talk about how to deal with your MTHFR. Methylation of folate is your key problem here. Not processing folate correctly leads to CHRONIC FATIGUE, never being able to make enough ATP. This is why you’re tired all the time, why

Let’s take a Quick Look at COMT, what it is, and how it affects patients. Is it linked to MTHFR? You betcha One of the base options for dealing with COMT is adenosyl/hydroxycobalamin, this mixture to two versions of B12 to support these

MTHFR, MTRR, MTR, ACHY, and COMT. These are the big 5 genetic SNPs that Gary Brecka and team look at. Its a great start but there are SO MANY more genes to look at to get the full picture. PEMT, GAD, SOD, NAT to name just a couple more. Y

This coming Sunday night at 7PM MST I discuss MTHFR (and other methylation errors), COMT, GAD1, FOLR2, and SOD errors during my Facebook Live. Remember that my book, THE 85% SOLUTION was on Amazon for 2 years regarding these subjects. But

Supports enzymes involved in processing sulfites and other compounds.

GGH encodes gamma-glutamyl hydrolase, which removes glutamate residues from folate compounds. This affects folate handling and helps explain why folate metabolism includes retention and processing as well as intake.

Makes an enzyme that helps produce a form of folate used in the processing of homocysteine. Common variants are different from rare variants that cause severe MTHFR deficiency.